크리스탈 캣츠

예전에 올렸던 글(http://www.crystalcats.net/tt/442)의 답글에서 견과류에 대해 이야기를 나누게 된 이후로 가끔씩 생각날 때마다 견과류의 부작용에 대한 정보를 검색해왔었습니다. 하지만, 자료 참 없더군요.

 독성있는 식물 리스트는 참 많지만, 경험상 그다지 믿을게 못된다고 생각하고 있습니다. 어떤 독성 성분이 조금 들어있다는 이유많으로 그 성분이 부작용을 일으키기까지는 얼마나 섭취해야 하는지, 실제로 부작용이 나타난 사례가 있었는지 같은 구체적인 내용을 전혀 밝히지 않은체 먹어서는 안되는 식물로 단정짓는 경우가 대부분이었기 때문입니다. 그래서 자세히 알아보면 결국 별거 아닌 것으로 밝혀지는 경우가 대부분이었죠.
이번에 다시 찾아본 목록들에도 마늘은 물론 알팔파, 알로에 처럼 허브요법에서 사용하는 것들 까지도 먹어서는 안되는 식물로 분류해  

Macadamia Nut

Common Name: Macadamia Nut
Scientific Name: Macadamia integrifolia
Family: Proteaceae
Toxic Principle: unknown
Clinical signs: depression, hyperthermia, weakness, muscular stiffness, vomiting, tremors, increased heartrate. Only reported in dogs at this time.



땅콩:peanut
잣:pinenut
호두:walnut
아몬드:almond
캐슈너트 [cashew nut]
밤 chestnut
은행 gingko nut
피스타치오 [pistachio]
마카다미아 [macadamia]


http://www.aspca.org/site/PageServer?pagename=pro_apcc_nontoxicplants

http://www.ncbi.nlm.nih.gov/sites/entrez?Db=pubmed&Cmd=ShowDetailView&TermToSearch=10670081&ordinalpos=3&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum

Weakness, tremors, and depression associated with macadamia nuts in dogs.

ASPCA National Animal Poison Control Center, Urbana, IL 61802, USA.

The ASPCA National Animal Poison Center managed 29 cases of ingestion of commercially available macadamia nuts in dogs during a 5-y period. Clinical signs included, from most to least, weakness, depression, vomiting, ataxia, tremor, hyperthermia, abdominal pain, lameness, stiffness, recumbency, and pale mucous membranes. The onset of clinical signs was reported as < 12 h in 79% of the cases. The duration of clinical signs for the majority of cases was < 24 h. The amount of macadamia nuts ingested was estimated in 72% of the calls with a mean of 11.7 g/kg bw. In an attempt to reproduce the syndrome, 4 dogs were gavaged with 20 g macadamia nuts/kg bw in a water slurry. The experimentally dosed dogs developed weakness, manifested by the inability to rise 12 h after dosing, mild central nervous system depression, vomiting, and hyperthermia, with rectal temperatures up to 40.5 C. Mild elevations in serum triglycerides and serum alkaline phosphatase were detected. Lipase values peaked sharply at 24 h and returned to normal by 48 h after dosing. Other serum biochemical and electrolyte determinations were unremarkable. Serum lipoprotein electrophoresis determinations were unchanged from baseline. The mechanism of the syndrome is unknown. All field and experimental dogs recovered uneventfully within 1 to 2 d whether treated by a veterinarian or not.